Microbiome Friendly Skincare
We are living in an age of technical innovation, particularly in the medical sciences. At Columbia SkinCare, we utilize the latest understanding of the skin microbiome to develop products that treat skincare as a health issue and not simply a cosmetic makeover.
Research on the human skin microbiome has advanced from a fledgling field to a flourishing area of medical research due to the promising results anticipated from this research. In addition to human health, microbiome research provides a foundation for a much broader scope of applications, as they offer a more sustainable use of environmental resources, as well as improve our food processing.
About your skin
The skin is colonized by a diverse community of microbes including bacteria, fungi, and viruses collectively called the skin microbiome. These commensal microbes play a key role in skin barrier function, educating the immune system, and protecting us from pathogens while contributing to healthy-looking skin. There is high variation within and between people in the diversity, composition, and function of the skin microbiome depending on body site, age, gender, and lifestyle factors.
ImProves KeratInocyte cohesIon
Summary
Our Probiotic Complex and Concentrate is an effective approach towards skin renewal. Recommended use will help to:
- induce production of key ingredients of skin quality and differentiation
- improve keratinocyte cohesion
- increase the production of antimicrobial peptides
- induce skin renewal and accelerate skin barrier recovery
In addition, environment plays a large role in the health and balance of our skin microbiome. Despite the rigors of daily hygiene, UV exposure, human, and animal contact, the facial skin microbiome is amazingly robust. The great majority of our facial skin microbes reside in the follicles and pores which act as personal microbial reservoirs allowing the skin surface to be repopulated quickly after cleansing or other agitations.
Increases the ProductIon of antImIcrobIal PePtIdes
Influence on barrier function
It is becoming clear to us that the transition from a healthy to disease microbiome may not involve colonization with new transient microbes, rather, disease flares likely involve expression of virulence genes in residence. For example, the commensal skin microbes are generally stable, and beneficial, but some have a dual “Jekyll and Hyde” personality. The factors that trigger the transition and the mechanisms of bacterial virulence are attractive targets for preventing and treating skin inflammation and maintaining a stable healthy skin microbiome.